Alzheimer's Disease explore - A Simple, Easy Way to Avoid Alzheimer's Disease

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For a long time the mechanisms through which some population contracted Alzheimer's disease Ad) was a mystery. They knew that there was some kind of genetic link because it tended to run in families, but beyond that, no one knew.

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How is Alzheimer's Disease explore - A Simple, Easy Way to Avoid Alzheimer's Disease

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Now, Alzheimer's disease study has made it potential to paint a much clearer picture. We now know that there are both genetic and environmental factors involved. We also know that the precursors to the disease begin appearing decades before Alzheimer's symptoms come to be evident, much like Hiv appears years before Aids develops.

We don't yet have as clear a ticket for the beginnings of Ad as Hiv is to Aids, but Alzheimer's disease study is working on it. Researchers are currently pursuing and validating several promising precursor markers. They also continue to identify and by comparison various environmental factors that play a part.

There are both inescapable indicators that identify elements connected with those who do not get Alzheimer's and negative indicators connected with those that do. For Alzheimer's disease prevention then, it would seem that all we have to do is use and adopt the inescapable and avoid the negative factors.

An example of a negative indicator could be major tooth loss. population who have lost more than 1/3 of their teeth are at a significantly higher risk for Alzheimer's and other dementias than those who have retained most of their teeth. So we brush and floss, visit a dentist regularly, take care of our teeth and keep them.

There are many inescapable factors, too many to go into here, but a good, strong example would be by maintaining the level of Vitamin B-12 in your bloodstream at 600 to 2,000 pg/ml. [In the U.S. The averages are generally much lower. Japan's "normal" range is 500-1,300 which may by comparison why Japan has such a low rate of Ad.]

By way of background, mammals, including humans, are born with serum levels of vitamin B12 at about 2,000 pg/ml (picograms--i.e., trillionths of a gram, per milliliter). The level declines throughout human life for various reasons, some known, others guessed at, many unknown.

Dr. John V. Dommisse, who practices treatment in Tucson, Arizona, wrote an article in 1991 in curative Hypotheses in which he maintained that most cases of Alzheimer's dementia are precisely missed B12 insufficiency cases. He later reported that Alzheimer's disease appeared to ensue from too-low serum vitamin B12.

Replenishing the vitamin succeeded in improving patients in spite of other risk factors. Repleting B12, agreeing to Dommisse, can reverse 75% of B12 insufficiency dementias when the health is discovered early enough.

More new study has put a finer point on it. For example, in Preventive treatment update (May 1995), Jeffrey Bland, PhD reported, "...a wide range of very foremost clinical outcomes from improving folate/B12/B6 status, cutting across neurological and arterial functional status."

Which ties in to the correlation between high levels of homocysteine and Ad. Reducing your level of homocysteine also makes you less likely to get Ad. Folates and other B-vitamins will sell out your homocysteine levels rapidly and economically. B-12 is cheap, available in a whole of forms.(See below for more on this.)

So, to summarize that area of Alzheimer's disease research, the bottom line is, a uncomplicated but sufficient step for Alzhemer's prevention is to get your levels of B-12 and homocysteine checked. Then take B-12, folates and other B-vitamins to raise your serum B-12 and lower your homocystein, to positive, anti-Ad ranges.

For more about Alzheimer's disease study and about Alzheimer's prevention, ensue the links below...

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